Minić, Arsa J.

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  • Minić, Arsa J. (2)
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Author's Bibliography

Mast cells in periapical lesions: potential role in their pathogenesis

Dražić, Radojica; Sopta, Jelena; Minić, Arsa J.

(Wiley-Blackwell Publishing, Inc, Malden, 2010)

TY  - JOUR
AU  - Dražić, Radojica
AU  - Sopta, Jelena
AU  - Minić, Arsa J.
PY  - 2010
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1521
AB  - Objective: The aim of this study was to qualitatively and semi-quantitatively analyze mast cells in periapical lesions. Materials and methods: Biopsy specimens of 96 periapical lesions were stained with hematoxylin-eosin, histochemical Giemsa and immunohistochemical CD 117 (C kit) antibody. Mast cell count below 100 mast cells on 1000 fields of high power magnification was noted as 'negative', 101-400 as 'mild', 401-800 cells as 'moderate', and over 800 cells as 'severe'. Results: Mast cells are found in 68 (70.8%) lesions. The presence of mast cells was greater in cysts than in granulomas (P  lt  0.0028). There was no difference in semi-quantitative expression of CD 117 in granulomas and cysts (P > 0.05). Mast cells were placed in both: inflammatory infiltrate and in fibroblastic areas of periapical lesions, and their presence was most frequently mild to moderate. Conclusions: The findings of present study could suggest a role of mast cells in regulation of cellular immune mechanisms in periapical lesions, balancing between alterative and reparatory processes in inflamed periapical tissue.
PB  - Wiley-Blackwell Publishing, Inc, Malden
T2  - Journal of Oral Pathology & Medicine
T1  - Mast cells in periapical lesions: potential role in their pathogenesis
VL  - 39
IS  - 3
SP  - 257
EP  - 262
DO  - 10.1111/j.1600-0714.2009.00870.x
ER  - 
@article{
author = "Dražić, Radojica and Sopta, Jelena and Minić, Arsa J.",
year = "2010",
abstract = "Objective: The aim of this study was to qualitatively and semi-quantitatively analyze mast cells in periapical lesions. Materials and methods: Biopsy specimens of 96 periapical lesions were stained with hematoxylin-eosin, histochemical Giemsa and immunohistochemical CD 117 (C kit) antibody. Mast cell count below 100 mast cells on 1000 fields of high power magnification was noted as 'negative', 101-400 as 'mild', 401-800 cells as 'moderate', and over 800 cells as 'severe'. Results: Mast cells are found in 68 (70.8%) lesions. The presence of mast cells was greater in cysts than in granulomas (P  lt  0.0028). There was no difference in semi-quantitative expression of CD 117 in granulomas and cysts (P > 0.05). Mast cells were placed in both: inflammatory infiltrate and in fibroblastic areas of periapical lesions, and their presence was most frequently mild to moderate. Conclusions: The findings of present study could suggest a role of mast cells in regulation of cellular immune mechanisms in periapical lesions, balancing between alterative and reparatory processes in inflamed periapical tissue.",
publisher = "Wiley-Blackwell Publishing, Inc, Malden",
journal = "Journal of Oral Pathology & Medicine",
title = "Mast cells in periapical lesions: potential role in their pathogenesis",
volume = "39",
number = "3",
pages = "257-262",
doi = "10.1111/j.1600-0714.2009.00870.x"
}
Dražić, R., Sopta, J.,& Minić, A. J.. (2010). Mast cells in periapical lesions: potential role in their pathogenesis. in Journal of Oral Pathology & Medicine
Wiley-Blackwell Publishing, Inc, Malden., 39(3), 257-262.
https://doi.org/10.1111/j.1600-0714.2009.00870.x
Dražić R, Sopta J, Minić AJ. Mast cells in periapical lesions: potential role in their pathogenesis. in Journal of Oral Pathology & Medicine. 2010;39(3):257-262.
doi:10.1111/j.1600-0714.2009.00870.x .
Dražić, Radojica, Sopta, Jelena, Minić, Arsa J., "Mast cells in periapical lesions: potential role in their pathogenesis" in Journal of Oral Pathology & Medicine, 39, no. 3 (2010):257-262,
https://doi.org/10.1111/j.1600-0714.2009.00870.x . .
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Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma

Dražić, Radojica; Minić, Arsa J.

(Mosby-Elsevier, New York, 1999)

TY  - JOUR
AU  - Dražić, Radojica
AU  - Minić, Arsa J.
PY  - 1999
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1106
AB  - A case of focal cemento-osseous dysplasia of the maxilla in a 19-year-old man is reported. Clinically, the lesion resembled periapical pathosis of odontogenic origin. The clinical and histopathologic features of cemento-osseous dysplasia are reviewed.
PB  - Mosby-Elsevier, New York
T2  - Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontology
T1  - Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma
VL  - 88
IS  - 1
SP  - 87
EP  - 89
DO  - 10.1016/S1079-2104(99)70198-8
ER  - 
@article{
author = "Dražić, Radojica and Minić, Arsa J.",
year = "1999",
abstract = "A case of focal cemento-osseous dysplasia of the maxilla in a 19-year-old man is reported. Clinically, the lesion resembled periapical pathosis of odontogenic origin. The clinical and histopathologic features of cemento-osseous dysplasia are reviewed.",
publisher = "Mosby-Elsevier, New York",
journal = "Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontology",
title = "Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma",
volume = "88",
number = "1",
pages = "87-89",
doi = "10.1016/S1079-2104(99)70198-8"
}
Dražić, R.,& Minić, A. J.. (1999). Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma. in Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontology
Mosby-Elsevier, New York., 88(1), 87-89.
https://doi.org/10.1016/S1079-2104(99)70198-8
Dražić R, Minić AJ. Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma. in Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontology. 1999;88(1):87-89.
doi:10.1016/S1079-2104(99)70198-8 .
Dražić, Radojica, Minić, Arsa J., "Focal cemento-osseous dysplasia in the maxilla mimicking periapical granuloma" in Oral Surgery, Oral Medicine, Oral Pathology, Oral Radiology & Endodontology, 88, no. 1 (1999):87-89,
https://doi.org/10.1016/S1079-2104(99)70198-8 . .
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