TY  - JOUR
AU  - Andrić, Miroslav
AU  - Dožić, Branko
AU  - Popović, Branka
AU  - Stefanović, D.
AU  - Basta-Jovanović, Gordana
AU  - Đogo, N.
AU  - Andjus, P.
AU  - Milašin, Jelena
PY  - 2010
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1531
AB  - Objectives: The aim of this study was to investigate the expression of survivin, an inhibitor of apoptosis, in odontogenic keratocysts and to compare it to the findings in non-neoplastic jaw cysts - periapical cysts, as well as to establish a possible relationship between survivin expression and human cytomegalovirus presence within these cysts. Materials and methods: Samples of 10 odontogenic keratocysts (five positive and five negative for the presence of cytomegalovirus, as determined by polymerase chain reaction) and 10 periapical cysts (five positive and five negative for the cytomegalovirus presence) were analysed. The expression of survivin was assessed by immunohistochemical methods, using monoclonal antibody that selectively recognizes the cytoplasmic form of survivin. Results: All 10 odontogenic keratocysts showed immunostaining for survivin, while all 10 periapical cysts were negative for its presence. There was no correlation between cytomegalovirus presence and expression of survivin within odontogenic keratocysts. Conclusion: Survivin may contribute to the aggressive behavior of odontogenic keratocysts, and thus support the emerging opinion of their neoplastic nature.
PB  - Wiley, Hoboken
T2  - Oral Diseases
T1  - Survivin expression in odontogenic keratocysts and correlation with cytomegalovirus infection
VL  - 16
IS  - 2
SP  - 156
EP  - 159
DO  - 10.1111/j.1601-0825.2009.01612.x
ER  - 

@article{
author = "Andrić, Miroslav and Dožić, Branko and Popović, Branka and Stefanović, D. and Basta-Jovanović, Gordana and Đogo, N. and Andjus, P. and Milašin, Jelena",
year = "2010",
abstract = "Objectives: The aim of this study was to investigate the expression of survivin, an inhibitor of apoptosis, in odontogenic keratocysts and to compare it to the findings in non-neoplastic jaw cysts - periapical cysts, as well as to establish a possible relationship between survivin expression and human cytomegalovirus presence within these cysts. Materials and methods: Samples of 10 odontogenic keratocysts (five positive and five negative for the presence of cytomegalovirus, as determined by polymerase chain reaction) and 10 periapical cysts (five positive and five negative for the cytomegalovirus presence) were analysed. The expression of survivin was assessed by immunohistochemical methods, using monoclonal antibody that selectively recognizes the cytoplasmic form of survivin. Results: All 10 odontogenic keratocysts showed immunostaining for survivin, while all 10 periapical cysts were negative for its presence. There was no correlation between cytomegalovirus presence and expression of survivin within odontogenic keratocysts. Conclusion: Survivin may contribute to the aggressive behavior of odontogenic keratocysts, and thus support the emerging opinion of their neoplastic nature.",
publisher = "Wiley, Hoboken",
journal = "Oral Diseases",
title = "Survivin expression in odontogenic keratocysts and correlation with cytomegalovirus infection",
volume = "16",
number = "2",
pages = "156-159",
doi = "10.1111/j.1601-0825.2009.01612.x"
}

Andrić, M., Dožić, B., Popović, B., Stefanović, D., Basta-Jovanović, G., Đogo, N., Andjus, P.,& Milašin, J.. (2010). Survivin expression in odontogenic keratocysts and correlation with cytomegalovirus infection. in Oral Diseases
Wiley, Hoboken., 16(2), 156-159.
https://doi.org/10.1111/j.1601-0825.2009.01612.x

Andrić M, Dožić B, Popović B, Stefanović D, Basta-Jovanović G, Đogo N, Andjus P, Milašin J. Survivin expression in odontogenic keratocysts and correlation with cytomegalovirus infection. in Oral Diseases. 2010;16(2):156-159.
doi:10.1111/j.1601-0825.2009.01612.x .

Andrić, Miroslav, Dožić, Branko, Popović, Branka, Stefanović, D., Basta-Jovanović, Gordana, Đogo, N., Andjus, P., Milašin, Jelena, "Survivin expression in odontogenic keratocysts and correlation with cytomegalovirus infection" in Oral Diseases, 16, no. 2 (2010):156-159,
https://doi.org/10.1111/j.1601-0825.2009.01612.x . .

TY  - JOUR
AU  - Novaković, I.
AU  - Bojić, D
AU  - Todorović, S
AU  - Apostolski, Slobodan
AU  - Luković, L
AU  - Stefanović, D
AU  - Milašin, Jelena
PY  - 2005
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1251
AB  - Alterations in production of cytoskeletal protein dystrophin caused by in-frame gene, mutations lead to the Becker muscular dystrophy. In this study we analyzed genotype-phenotype correlation in a group of Becker musculaudystrophy patients with deletions affecting the proximal part of dystrophin gene, encompassing exons 3-13. Four patients with deletions affecting N terminal dystrophin domain had early onset and faster progression of the disease, while three patients with deletions in the proximal part, of dystrophin's rod domain had a more benign disease course. Our study suggests that proximal gene deletions in Becker muscular dystrophy have various phenotypic effects depending on the affected domain of protein dystrophin.
PB  - New York Acad Sciences, New York
T2  - Biophysics from Molecules to Brain: in Memory of Radoslav K. Andjus
T1  - Proximal dystrophin gene deletions and protein alterations in Becker muscular dystrophy
VL  - 1048
SP  - 406
EP  - 410
DO  - 10.1196/annals.1342.050
ER  - 

@article{
author = "Novaković, I. and Bojić, D and Todorović, S and Apostolski, Slobodan and Luković, L and Stefanović, D and Milašin, Jelena",
year = "2005",
abstract = "Alterations in production of cytoskeletal protein dystrophin caused by in-frame gene, mutations lead to the Becker muscular dystrophy. In this study we analyzed genotype-phenotype correlation in a group of Becker musculaudystrophy patients with deletions affecting the proximal part of dystrophin gene, encompassing exons 3-13. Four patients with deletions affecting N terminal dystrophin domain had early onset and faster progression of the disease, while three patients with deletions in the proximal part, of dystrophin's rod domain had a more benign disease course. Our study suggests that proximal gene deletions in Becker muscular dystrophy have various phenotypic effects depending on the affected domain of protein dystrophin.",
publisher = "New York Acad Sciences, New York",
journal = "Biophysics from Molecules to Brain: in Memory of Radoslav K. Andjus",
title = "Proximal dystrophin gene deletions and protein alterations in Becker muscular dystrophy",
volume = "1048",
pages = "406-410",
doi = "10.1196/annals.1342.050"
}

Novaković, I., Bojić, D., Todorović, S., Apostolski, S., Luković, L., Stefanović, D.,& Milašin, J.. (2005). Proximal dystrophin gene deletions and protein alterations in Becker muscular dystrophy. in Biophysics from Molecules to Brain: in Memory of Radoslav K. Andjus
New York Acad Sciences, New York., 1048, 406-410.
https://doi.org/10.1196/annals.1342.050

Novaković I, Bojić D, Todorović S, Apostolski S, Luković L, Stefanović D, Milašin J. Proximal dystrophin gene deletions and protein alterations in Becker muscular dystrophy. in Biophysics from Molecules to Brain: in Memory of Radoslav K. Andjus. 2005;1048:406-410.
doi:10.1196/annals.1342.050 .

Novaković, I., Bojić, D, Todorović, S, Apostolski, Slobodan, Luković, L, Stefanović, D, Milašin, Jelena, "Proximal dystrophin gene deletions and protein alterations in Becker muscular dystrophy" in Biophysics from Molecules to Brain: in Memory of Radoslav K. Andjus, 1048 (2005):406-410,
https://doi.org/10.1196/annals.1342.050 . .