TY  - CONF
AU  - Jurišić, Vladimir
AU  - Terzić, T.
AU  - Jurišić, Milan
PY  - 2013
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1822
PB  - Academic Press Ltd- Elsevier Science Ltd, London
C3  - Cytokine
T1  - Values of TNF alpha in cystic fluid in odontogenic tumor depending on macrophage number in cystic wall
VL  - 63
IS  - 3
SP  - 274
EP  - 275
DO  - 10.1016/j.cyto.2013.06.138
ER  - 

@conference{
author = "Jurišić, Vladimir and Terzić, T. and Jurišić, Milan",
year = "2013",
publisher = "Academic Press Ltd- Elsevier Science Ltd, London",
journal = "Cytokine",
title = "Values of TNF alpha in cystic fluid in odontogenic tumor depending on macrophage number in cystic wall",
volume = "63",
number = "3",
pages = "274-275",
doi = "10.1016/j.cyto.2013.06.138"
}

Jurišić, V., Terzić, T.,& Jurišić, M.. (2013). Values of TNF alpha in cystic fluid in odontogenic tumor depending on macrophage number in cystic wall. in Cytokine
Academic Press Ltd- Elsevier Science Ltd, London., 63(3), 274-275.
https://doi.org/10.1016/j.cyto.2013.06.138

Jurišić V, Terzić T, Jurišić M. Values of TNF alpha in cystic fluid in odontogenic tumor depending on macrophage number in cystic wall. in Cytokine. 2013;63(3):274-275.
doi:10.1016/j.cyto.2013.06.138 .

Jurišić, Vladimir, Terzić, T., Jurišić, Milan, "Values of TNF alpha in cystic fluid in odontogenic tumor depending on macrophage number in cystic wall" in Cytokine, 63, no. 3 (2013):274-275,
https://doi.org/10.1016/j.cyto.2013.06.138 . .

TY  - JOUR
AU  - Jurišić, Milan
AU  - Andrić, Miroslav
AU  - dos Santos, J. N.
AU  - Jurišić, Vladimir
PY  - 2012
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1761
AB  - Keratocystic odontogenic tumors (KCOTs) are benign but locally aggressive lesions of the gnathic skeleton with high propensity to recur following surgical treatment. High proliferative activity of KCOTs epithelial cells is considered as one of the factors contributing to their aggressive clinical behavior Aggressive growth within the jaws, tendency to invade surrounding anatomical structures and occasional malignant alteration are the features that distinguish KCOTs from other types of odontogenic tumors. Due to their unique clinical and biological features, KCOTs still present an important problem in oral and maxillofacial surgery. This is especially true when a choice of the most appropriate treatment modality should be made. Establishing balance between effective reduction of recurrence risk and selection of a less aggressive surgical procedure is an issue that should be carefully considered for each individual patient.
PB  - Balkan Union of Oncology (B.U.ON.)
T2  - Journal of BUON
T1  - Clinical, diagnostic and therapeutic features of keratocystic odontogenic tumors: a review
VL  - 17
IS  - 2
SP  - 237
EP  - 244
UR  - https://hdl.handle.net/21.15107/rcub_smile_1761
ER  - 

@article{
author = "Jurišić, Milan and Andrić, Miroslav and dos Santos, J. N. and Jurišić, Vladimir",
year = "2012",
abstract = "Keratocystic odontogenic tumors (KCOTs) are benign but locally aggressive lesions of the gnathic skeleton with high propensity to recur following surgical treatment. High proliferative activity of KCOTs epithelial cells is considered as one of the factors contributing to their aggressive clinical behavior Aggressive growth within the jaws, tendency to invade surrounding anatomical structures and occasional malignant alteration are the features that distinguish KCOTs from other types of odontogenic tumors. Due to their unique clinical and biological features, KCOTs still present an important problem in oral and maxillofacial surgery. This is especially true when a choice of the most appropriate treatment modality should be made. Establishing balance between effective reduction of recurrence risk and selection of a less aggressive surgical procedure is an issue that should be carefully considered for each individual patient.",
publisher = "Balkan Union of Oncology (B.U.ON.)",
journal = "Journal of BUON",
title = "Clinical, diagnostic and therapeutic features of keratocystic odontogenic tumors: a review",
volume = "17",
number = "2",
pages = "237-244",
url = "https://hdl.handle.net/21.15107/rcub_smile_1761"
}

Jurišić, M., Andrić, M., dos Santos, J. N.,& Jurišić, V.. (2012). Clinical, diagnostic and therapeutic features of keratocystic odontogenic tumors: a review. in Journal of BUON
Balkan Union of Oncology (B.U.ON.)., 17(2), 237-244.
https://hdl.handle.net/21.15107/rcub_smile_1761

Jurišić M, Andrić M, dos Santos JN, Jurišić V. Clinical, diagnostic and therapeutic features of keratocystic odontogenic tumors: a review. in Journal of BUON. 2012;17(2):237-244.
https://hdl.handle.net/21.15107/rcub_smile_1761 .

Jurišić, Milan, Andrić, Miroslav, dos Santos, J. N., Jurišić, Vladimir, "Clinical, diagnostic and therapeutic features of keratocystic odontogenic tumors: a review" in Journal of BUON, 17, no. 2 (2012):237-244,
https://hdl.handle.net/21.15107/rcub_smile_1761 .

TY  - CONF
AU  - Jurišić, Vladimir
AU  - Čolić, Snježana
AU  - Jurišić, Milan
PY  - 2009
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1493
PB  - Springer Tokyo, Tokyo
C3  - Journal of Physiological Sciences
T1  - LDH and tnf-alpha in odontogenic keratocysts
VL  - 59
SP  - 530
EP  - 530
UR  - https://hdl.handle.net/21.15107/rcub_smile_1493
ER  - 

@conference{
author = "Jurišić, Vladimir and Čolić, Snježana and Jurišić, Milan",
year = "2009",
publisher = "Springer Tokyo, Tokyo",
journal = "Journal of Physiological Sciences",
title = "LDH and tnf-alpha in odontogenic keratocysts",
volume = "59",
pages = "530-530",
url = "https://hdl.handle.net/21.15107/rcub_smile_1493"
}

Jurišić, V., Čolić, S.,& Jurišić, M.. (2009). LDH and tnf-alpha in odontogenic keratocysts. in Journal of Physiological Sciences
Springer Tokyo, Tokyo., 59, 530-530.
https://hdl.handle.net/21.15107/rcub_smile_1493

Jurišić V, Čolić S, Jurišić M. LDH and tnf-alpha in odontogenic keratocysts. in Journal of Physiological Sciences. 2009;59:530-530.
https://hdl.handle.net/21.15107/rcub_smile_1493 .

Jurišić, Vladimir, Čolić, Snježana, Jurišić, Milan, "LDH and tnf-alpha in odontogenic keratocysts" in Journal of Physiological Sciences, 59 (2009):530-530,
https://hdl.handle.net/21.15107/rcub_smile_1493 .

TY  - JOUR
AU  - Jurišić, Vladimir
AU  - Terzić, T.
AU  - Čolić, Snježana
AU  - Jurišić, Milan
PY  - 2008
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1445
AB  - OBJECTIVE: To correlate values of tumor necrosis factor-alpha (TNF-alpha) depending on the count of inflammatory cells with degree of vascularization in cystic fluid of radicular cysts. MATERIAL AND METHODS: We investigated TNF-alpha concentration in 43 radicular cysts obtained from patients undergoing surgery, under local anaesthesia, and after aspiration of cystic fluid from non-ruptured cysts by enzyme-linked immunosorbent assay assay in respect of different clinical parameters as well as by histomorphometric analyses. RESULTS: Significantly higher concentration of TNF-alpha is associated with smaller radicular cysts, higher protein concentration in cystic fluid as well as with higher presence of inflammatory cells, and increased degree of vascularization in pericystic tissues and cyst wall thickness. CONCLUSIONS: We believe that determination of TNF-alpha in cystic fluid simultaneously with other parameters can be an additional parameter for clinical diagnosis of inflammed cysts.
PB  - Wiley, Hoboken
T2  - Oral Diseases
T1  - The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts
VL  - 14
IS  - 7
SP  - 600
EP  - 605
DO  - 10.1111/j.1601-0825.2007.01426.x
ER  - 

@article{
author = "Jurišić, Vladimir and Terzić, T. and Čolić, Snježana and Jurišić, Milan",
year = "2008",
abstract = "OBJECTIVE: To correlate values of tumor necrosis factor-alpha (TNF-alpha) depending on the count of inflammatory cells with degree of vascularization in cystic fluid of radicular cysts. MATERIAL AND METHODS: We investigated TNF-alpha concentration in 43 radicular cysts obtained from patients undergoing surgery, under local anaesthesia, and after aspiration of cystic fluid from non-ruptured cysts by enzyme-linked immunosorbent assay assay in respect of different clinical parameters as well as by histomorphometric analyses. RESULTS: Significantly higher concentration of TNF-alpha is associated with smaller radicular cysts, higher protein concentration in cystic fluid as well as with higher presence of inflammatory cells, and increased degree of vascularization in pericystic tissues and cyst wall thickness. CONCLUSIONS: We believe that determination of TNF-alpha in cystic fluid simultaneously with other parameters can be an additional parameter for clinical diagnosis of inflammed cysts.",
publisher = "Wiley, Hoboken",
journal = "Oral Diseases",
title = "The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts",
volume = "14",
number = "7",
pages = "600-605",
doi = "10.1111/j.1601-0825.2007.01426.x"
}

Jurišić, V., Terzić, T., Čolić, S.,& Jurišić, M.. (2008). The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts. in Oral Diseases
Wiley, Hoboken., 14(7), 600-605.
https://doi.org/10.1111/j.1601-0825.2007.01426.x

Jurišić V, Terzić T, Čolić S, Jurišić M. The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts. in Oral Diseases. 2008;14(7):600-605.
doi:10.1111/j.1601-0825.2007.01426.x .

Jurišić, Vladimir, Terzić, T., Čolić, Snježana, Jurišić, Milan, "The concentration of TNF-alpha correlate with number of inflammatory cells and degree of vascularization in radicular cysts" in Oral Diseases, 14, no. 7 (2008):600-605,
https://doi.org/10.1111/j.1601-0825.2007.01426.x . .

TY  - JOUR
AU  - Čolić, Snježana
AU  - Jurišić, Milan
AU  - Jurišić, Vladimir
PY  - 2008
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1398
AB  - The radicular cysts are result of inflammatory process in the periapical tissues associated with necrotic and infected pulps. Humoral and cellular immune responses play a central role in the pathogenesis of these lesions. The most important role in the growth of these lesion have proinflammatore cytokine TNF-α, IL-1 and IL-6. Cytokine can be secreted by macrophages, monocytes and other cells of the immune system and can participate in skeletal homeostasis including osteoclastic formation, and bone resorption in maxillofacial region. The aim of this study is to give a concise review for mechanism of growth of maxilofacial radicular cysts, indicated of clinical aspect, as well as explained role of cytokine in this pathophysiology process.
AB  - Radikularne ciste su inflamatorne patološke lezije koje se razvijaju na vrhu korena avitalnog zuba cističnom transformacijom prethodno formiranog epitelnog granuloma. Humoralni i celularni imunitet imaju važnu ulogu u mehanizmu nastanka ovih lezija. Najvažniji medijatori u razvoju periapikalnih lezija su proinflamatorni citokini TNF- α (faktor nekroze tumora-α), IL-1(interleukin-1) i IL- 6 (Interleukin-6). Proizvode ih kako odbrambene ćelije, tako i ćelije lokalnog tkiva u različitom stepenu. Svi ovi citokini učestvuju u koštanoj homeostazi, uključ ujući i koštanu resorpciju u maksilofacijalnom skeletu. Cilj ovog rada je da pokaže osnovne mehanizme nastanka radikularnih cista vilice, ukaže na klinič ki značaj kao da se pojasni ulogu citokina u patofiziološ kim procesima.
PB  - Udruženje hirurga Jugoslavije, Beograd
T2  - Acta chirurgica iugoslavica
T1  - Pathophysiological mechanism of the developing maxillofacial radicular cyst
T1  - Patofiziološki mehanizmi nastanka radikularnih cista vilice
VL  - 55
IS  - 1
SP  - 87
EP  - 92
DO  - 10.2298/ACI0801087C
ER  - 

@article{
author = "Čolić, Snježana and Jurišić, Milan and Jurišić, Vladimir",
year = "2008",
abstract = "The radicular cysts are result of inflammatory process in the periapical tissues associated with necrotic and infected pulps. Humoral and cellular immune responses play a central role in the pathogenesis of these lesions. The most important role in the growth of these lesion have proinflammatore cytokine TNF-α, IL-1 and IL-6. Cytokine can be secreted by macrophages, monocytes and other cells of the immune system and can participate in skeletal homeostasis including osteoclastic formation, and bone resorption in maxillofacial region. The aim of this study is to give a concise review for mechanism of growth of maxilofacial radicular cysts, indicated of clinical aspect, as well as explained role of cytokine in this pathophysiology process., Radikularne ciste su inflamatorne patološke lezije koje se razvijaju na vrhu korena avitalnog zuba cističnom transformacijom prethodno formiranog epitelnog granuloma. Humoralni i celularni imunitet imaju važnu ulogu u mehanizmu nastanka ovih lezija. Najvažniji medijatori u razvoju periapikalnih lezija su proinflamatorni citokini TNF- α (faktor nekroze tumora-α), IL-1(interleukin-1) i IL- 6 (Interleukin-6). Proizvode ih kako odbrambene ćelije, tako i ćelije lokalnog tkiva u različitom stepenu. Svi ovi citokini učestvuju u koštanoj homeostazi, uključ ujući i koštanu resorpciju u maksilofacijalnom skeletu. Cilj ovog rada je da pokaže osnovne mehanizme nastanka radikularnih cista vilice, ukaže na klinič ki značaj kao da se pojasni ulogu citokina u patofiziološ kim procesima.",
publisher = "Udruženje hirurga Jugoslavije, Beograd",
journal = "Acta chirurgica iugoslavica",
title = "Pathophysiological mechanism of the developing maxillofacial radicular cyst, Patofiziološki mehanizmi nastanka radikularnih cista vilice",
volume = "55",
number = "1",
pages = "87-92",
doi = "10.2298/ACI0801087C"
}

Čolić, S., Jurišić, M.,& Jurišić, V.. (2008). Pathophysiological mechanism of the developing maxillofacial radicular cyst. in Acta chirurgica iugoslavica
Udruženje hirurga Jugoslavije, Beograd., 55(1), 87-92.
https://doi.org/10.2298/ACI0801087C

Čolić S, Jurišić M, Jurišić V. Pathophysiological mechanism of the developing maxillofacial radicular cyst. in Acta chirurgica iugoslavica. 2008;55(1):87-92.
doi:10.2298/ACI0801087C .

Čolić, Snježana, Jurišić, Milan, Jurišić, Vladimir, "Pathophysiological mechanism of the developing maxillofacial radicular cyst" in Acta chirurgica iugoslavica, 55, no. 1 (2008):87-92,
https://doi.org/10.2298/ACI0801087C . .

TY  - CONF
AU  - Jurišić, Vladimir
AU  - Čolić, Snježana
AU  - Terzić, Tatjana
AU  - Jurišić, Milan
PY  - 2008
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1389
PB  - Karger, Basel
C3  - Tumor Biology
T1  - NF-alpha in Radicular and Keratocyst of Maxilofacial Region
VL  - 29
SP  - 39
EP  - 39
UR  - https://hdl.handle.net/21.15107/rcub_smile_1389
ER  - 

@conference{
author = "Jurišić, Vladimir and Čolić, Snježana and Terzić, Tatjana and Jurišić, Milan",
year = "2008",
publisher = "Karger, Basel",
journal = "Tumor Biology",
title = "NF-alpha in Radicular and Keratocyst of Maxilofacial Region",
volume = "29",
pages = "39-39",
url = "https://hdl.handle.net/21.15107/rcub_smile_1389"
}

Jurišić, V., Čolić, S., Terzić, T.,& Jurišić, M.. (2008). NF-alpha in Radicular and Keratocyst of Maxilofacial Region. in Tumor Biology
Karger, Basel., 29, 39-39.
https://hdl.handle.net/21.15107/rcub_smile_1389

Jurišić V, Čolić S, Terzić T, Jurišić M. NF-alpha in Radicular and Keratocyst of Maxilofacial Region. in Tumor Biology. 2008;29:39-39.
https://hdl.handle.net/21.15107/rcub_smile_1389 .

Jurišić, Vladimir, Čolić, Snježana, Terzić, Tatjana, Jurišić, Milan, "NF-alpha in Radicular and Keratocyst of Maxilofacial Region" in Tumor Biology, 29 (2008):39-39,
https://hdl.handle.net/21.15107/rcub_smile_1389 .

TY  - JOUR
AU  - Popović, Branka
AU  - Jekić, B.
AU  - Novaković, I.
AU  - Luković, Ljiljana
AU  - Tepavčević, Zvezdana
AU  - Jurišić, Vladimir
AU  - Vukadinović, Miroslav
AU  - Milašin, Jelena
PY  - 2007
UR  - https://smile.stomf.bg.ac.rs/handle/123456789/1329
AB  - Apoptosis is a genetically regulated process involved in tissue size regulation, morphogenesis, and elimination of genetically damaged cells. A pallet of genes is involved in the control of apoptosis, such as bcl-2 family whose oncogenic potential has been demonstrated in oral tumorigenesis. Different members of bcl-2 family may promote or inhibit apoptosis by synthesizing anti- and proapoptotic proteins. One of antiapoptotic proteins, bcl-2, with a crucial role in apoptosis regulation was the object of our study. By means of immunohistochemistry we estimated the level of overexpression of bcl-2 proteins in a series of the 26 formalin fixed, paraffin-embedded samples of oral squamous cell carcinoma (OSCC). Analyzed tumors originated from different sites of oral cavity; 7/26 belonged to stage II, 14/26 to stage III, and 5/26 to stage IV Immunoreactivity was scored according to the percentage and intensity of positive cytoplasmic bcl-2 staining. All tumors had low percentage of positively stained bcl-2 cells, with mean values for lower/higher intensity of 8.3 +/- 2.5/34.4 +/- 7, 7.5 +/- 1.1/31.9 +/- 4.3, and 8.4 +/- 5.8/31.5 +/- 5.8 within stages II,III, and IV, respectively. Low level of bcl-2 expression in our sample seems to be associated with higher survival rate: 77% for the 5-year follow-up period. Comparing clinicopathologic and risk factors data within each and between three groups of analyzed tumors (lip-tongue P = 0.58, tongue-floor of the mouth, P = 0.21, lip-floor of the mouth, P = 0.50) there was no significant difference. However, [sic].
PB  - Wiley-Blackwell, Hoboken
T2  - Signal Transduction Pathways, Pt C: Cell Signaling in Health & Disease
T1  - Bcl-2 expression in oral squamous cell carcinoma
VL  - 1095
SP  - 19
EP  - 25
DO  - 10.1196/annals.1397.003
ER  - 

@article{
author = "Popović, Branka and Jekić, B. and Novaković, I. and Luković, Ljiljana and Tepavčević, Zvezdana and Jurišić, Vladimir and Vukadinović, Miroslav and Milašin, Jelena",
year = "2007",
abstract = "Apoptosis is a genetically regulated process involved in tissue size regulation, morphogenesis, and elimination of genetically damaged cells. A pallet of genes is involved in the control of apoptosis, such as bcl-2 family whose oncogenic potential has been demonstrated in oral tumorigenesis. Different members of bcl-2 family may promote or inhibit apoptosis by synthesizing anti- and proapoptotic proteins. One of antiapoptotic proteins, bcl-2, with a crucial role in apoptosis regulation was the object of our study. By means of immunohistochemistry we estimated the level of overexpression of bcl-2 proteins in a series of the 26 formalin fixed, paraffin-embedded samples of oral squamous cell carcinoma (OSCC). Analyzed tumors originated from different sites of oral cavity; 7/26 belonged to stage II, 14/26 to stage III, and 5/26 to stage IV Immunoreactivity was scored according to the percentage and intensity of positive cytoplasmic bcl-2 staining. All tumors had low percentage of positively stained bcl-2 cells, with mean values for lower/higher intensity of 8.3 +/- 2.5/34.4 +/- 7, 7.5 +/- 1.1/31.9 +/- 4.3, and 8.4 +/- 5.8/31.5 +/- 5.8 within stages II,III, and IV, respectively. Low level of bcl-2 expression in our sample seems to be associated with higher survival rate: 77% for the 5-year follow-up period. Comparing clinicopathologic and risk factors data within each and between three groups of analyzed tumors (lip-tongue P = 0.58, tongue-floor of the mouth, P = 0.21, lip-floor of the mouth, P = 0.50) there was no significant difference. However, [sic].",
publisher = "Wiley-Blackwell, Hoboken",
journal = "Signal Transduction Pathways, Pt C: Cell Signaling in Health & Disease",
title = "Bcl-2 expression in oral squamous cell carcinoma",
volume = "1095",
pages = "19-25",
doi = "10.1196/annals.1397.003"
}

Popović, B., Jekić, B., Novaković, I., Luković, L., Tepavčević, Z., Jurišić, V., Vukadinović, M.,& Milašin, J.. (2007). Bcl-2 expression in oral squamous cell carcinoma. in Signal Transduction Pathways, Pt C: Cell Signaling in Health & Disease
Wiley-Blackwell, Hoboken., 1095, 19-25.
https://doi.org/10.1196/annals.1397.003

Popović B, Jekić B, Novaković I, Luković L, Tepavčević Z, Jurišić V, Vukadinović M, Milašin J. Bcl-2 expression in oral squamous cell carcinoma. in Signal Transduction Pathways, Pt C: Cell Signaling in Health & Disease. 2007;1095:19-25.
doi:10.1196/annals.1397.003 .

Popović, Branka, Jekić, B., Novaković, I., Luković, Ljiljana, Tepavčević, Zvezdana, Jurišić, Vladimir, Vukadinović, Miroslav, Milašin, Jelena, "Bcl-2 expression in oral squamous cell carcinoma" in Signal Transduction Pathways, Pt C: Cell Signaling in Health & Disease, 1095 (2007):19-25,
https://doi.org/10.1196/annals.1397.003 . .