SMILE – Repository of the Faculty of Dental Medicine
University of Belgrade - Faculty of Dental Medicine
    • English
    • Српски
    • Српски (Serbia)
  • English 
    • English
    • Serbian (Cyrilic)
    • Serbian (Latin)
  • Login
View Item 
  •   SMILE
  • Stomatološki fakultet
  • Radovi istraživača
  • View Item
  •   SMILE
  • Stomatološki fakultet
  • Radovi istraživača
  • View Item
JavaScript is disabled for your browser. Some features of this site may not work without it.

Pathophysiological mechanism of the developing maxillofacial radicular cyst

Patofiziološki mehanizmi nastanka radikularnih cista vilice

Thumbnail
2008
1393.pdf (535.2Kb)
Authors
Čolić, Snježana
Jurišić, Milan
Jurišić, Vladimir
Article (Published version)
Metadata
Show full item record
Abstract
The radicular cysts are result of inflammatory process in the periapical tissues associated with necrotic and infected pulps. Humoral and cellular immune responses play a central role in the pathogenesis of these lesions. The most important role in the growth of these lesion have proinflammatore cytokine TNF-α, IL-1 and IL-6. Cytokine can be secreted by macrophages, monocytes and other cells of the immune system and can participate in skeletal homeostasis including osteoclastic formation, and bone resorption in maxillofacial region. The aim of this study is to give a concise review for mechanism of growth of maxilofacial radicular cysts, indicated of clinical aspect, as well as explained role of cytokine in this pathophysiology process.
Radikularne ciste su inflamatorne patološke lezije koje se razvijaju na vrhu korena avitalnog zuba cističnom transformacijom prethodno formiranog epitelnog granuloma. Humoralni i celularni imunitet imaju važnu ulogu u mehanizmu nastanka ovih lezija. Najvažniji medijatori u razvoju periapikalnih lezija su proinflamatorni citokini TNF- α (faktor nekroze tumora-α), IL-1(interleukin-1) i IL- 6 (Interleukin-6). Proizvode ih kako odbrambene ćelije, tako i ćelije lokalnog tkiva u različitom stepenu. Svi ovi citokini učestvuju u koštanoj homeostazi, uključ ujući i koštanu resorpciju u maksilofacijalnom skeletu. Cilj ovog rada je da pokaže osnovne mehanizme nastanka radikularnih cista vilice, ukaže na klinič ki značaj kao da se pojasni ulogu citokina u patofiziološ kim procesima.
Keywords:
radicular cysts / epidemiology / histology / therapy / immunology / clinical aspect / cytokine / radikularne ciste / epidemiologija / histologija / imunologija / citokini
Source:
Acta chirurgica iugoslavica, 2008, 55, 1, 87-92
Publisher:
  • Udruženje hirurga Jugoslavije, Beograd

DOI: 10.2298/ACI0801087C

ISSN: 0354-950X

Scopus: 2-s2.0-45749133156
[ Google Scholar ]
4
URI
http://smile.stomf.bg.ac.rs/handle/123456789/1398
Collections
  • Radovi istraživača
Institution
Stomatološki fakultet
TY  - JOUR
AU  - Čolić, Snježana
AU  - Jurišić, Milan
AU  - Jurišić, Vladimir
PY  - 2008
UR  - http://smile.stomf.bg.ac.rs/handle/123456789/1398
AB  - The radicular cysts are result of inflammatory process in the periapical tissues associated with necrotic and infected pulps. Humoral and cellular immune responses play a central role in the pathogenesis of these lesions. The most important role in the growth of these lesion have proinflammatore cytokine TNF-α, IL-1 and IL-6. Cytokine can be secreted by macrophages, monocytes and other cells of the immune system and can participate in skeletal homeostasis including osteoclastic formation, and bone resorption in maxillofacial region. The aim of this study is to give a concise review for mechanism of growth of maxilofacial radicular cysts, indicated of clinical aspect, as well as explained role of cytokine in this pathophysiology process.
AB  - Radikularne ciste su inflamatorne patološke lezije koje se razvijaju na vrhu korena avitalnog zuba cističnom transformacijom prethodno formiranog epitelnog granuloma. Humoralni i celularni imunitet imaju važnu ulogu u mehanizmu nastanka ovih lezija. Najvažniji medijatori u razvoju periapikalnih lezija su proinflamatorni citokini TNF- α (faktor nekroze tumora-α), IL-1(interleukin-1) i IL- 6 (Interleukin-6). Proizvode ih kako odbrambene ćelije, tako i ćelije lokalnog tkiva u različitom stepenu. Svi ovi citokini učestvuju u koštanoj homeostazi, uključ ujući i koštanu resorpciju u maksilofacijalnom skeletu. Cilj ovog rada je da pokaže osnovne mehanizme nastanka radikularnih cista vilice, ukaže na klinič ki značaj kao da se pojasni ulogu citokina u patofiziološ kim procesima.
PB  - Udruženje hirurga Jugoslavije, Beograd
C3  - Acta chirurgica iugoslavica
T1  - Pathophysiological mechanism of the developing maxillofacial radicular cyst
T1  - Patofiziološki mehanizmi nastanka radikularnih cista vilice
VL  - 55
IS  - 1
SP  - 87
EP  - 92
DO  - 10.2298/ACI0801087C
ER  - 
@article{
author = "Čolić, Snježana and Jurišić, Milan and Jurišić, Vladimir",
year = "2008",
url = "http://smile.stomf.bg.ac.rs/handle/123456789/1398",
abstract = "The radicular cysts are result of inflammatory process in the periapical tissues associated with necrotic and infected pulps. Humoral and cellular immune responses play a central role in the pathogenesis of these lesions. The most important role in the growth of these lesion have proinflammatore cytokine TNF-α, IL-1 and IL-6. Cytokine can be secreted by macrophages, monocytes and other cells of the immune system and can participate in skeletal homeostasis including osteoclastic formation, and bone resorption in maxillofacial region. The aim of this study is to give a concise review for mechanism of growth of maxilofacial radicular cysts, indicated of clinical aspect, as well as explained role of cytokine in this pathophysiology process., Radikularne ciste su inflamatorne patološke lezije koje se razvijaju na vrhu korena avitalnog zuba cističnom transformacijom prethodno formiranog epitelnog granuloma. Humoralni i celularni imunitet imaju važnu ulogu u mehanizmu nastanka ovih lezija. Najvažniji medijatori u razvoju periapikalnih lezija su proinflamatorni citokini TNF- α (faktor nekroze tumora-α), IL-1(interleukin-1) i IL- 6 (Interleukin-6). Proizvode ih kako odbrambene ćelije, tako i ćelije lokalnog tkiva u različitom stepenu. Svi ovi citokini učestvuju u koštanoj homeostazi, uključ ujući i koštanu resorpciju u maksilofacijalnom skeletu. Cilj ovog rada je da pokaže osnovne mehanizme nastanka radikularnih cista vilice, ukaže na klinič ki značaj kao da se pojasni ulogu citokina u patofiziološ kim procesima.",
publisher = "Udruženje hirurga Jugoslavije, Beograd",
journal = "Acta chirurgica iugoslavica",
title = "Pathophysiological mechanism of the developing maxillofacial radicular cyst, Patofiziološki mehanizmi nastanka radikularnih cista vilice",
volume = "55",
number = "1",
pages = "87-92",
doi = "10.2298/ACI0801087C"
}
Čolić S, Jurišić M, Jurišić V. Patofiziološki mehanizmi nastanka radikularnih cista vilice. Acta chirurgica iugoslavica. 2008;55(1):87-92
Čolić, S., Jurišić, M.,& Jurišić, V. (2008). Patofiziološki mehanizmi nastanka radikularnih cista vilice.
Acta chirurgica iugoslavicaUdruženje hirurga Jugoslavije, Beograd., 55(1), 87-92.
https://doi.org/10.2298/ACI0801087C
Čolić Snježana, Jurišić Milan, Jurišić Vladimir, "Patofiziološki mehanizmi nastanka radikularnih cista vilice" 55, no. 1 (2008):87-92,
https://doi.org/10.2298/ACI0801087C .

DSpace software copyright © 2002-2015  DuraSpace
About Smile – School of dental Medicine dIgitaL archivE | Send Feedback

OpenAIRERCUB
 

 

All of DSpaceInstitutionsAuthorsTitlesSubjectsThis institutionAuthorsTitlesSubjects

Statistics

View Usage Statistics

DSpace software copyright © 2002-2015  DuraSpace
About Smile – School of dental Medicine dIgitaL archivE | Send Feedback

OpenAIRERCUB